Parkinsons Disease Essay -- Medical Medical Medicine Essays Treatment

Parkinsons disorderParkinsons ingrainion (PD), the shaking palsy first described by James Parkinson in 1817, is a progressive neurodegenerative disorder which alludes in up of 1.5 million Americans. The disease begins to occur around age 40 and has relative relative incidence with patient age. One survey found that PD whitethorn affect 1% of the population over 60. Incidence seems to be more turgid in men, and tends to progress to incapacity and death over one or two decades. Clinical diagnosis of PD is truely solely myrmecophilous on the presentation of the symptoms by the patient which reflect a wishing of striatal dopamine causal agentd by the destruction of the cells in the substantia nigra. Imaging and other research lab techniques can be used to rule out other disorders, nevertheless are not necessary for the actual diagnosis of PD. The first press of PA is usually bradykinesia. Movements are usually quite slow. Routine activities whitethorn require deliberate planning and thought for execution. Difficulty initiating movements or akinesia, may besides be present. Rigidity in the flexors is to a fault present. This is due to an magnified response to normal proprioceptive return from the somatic musculature. A resting dismay of 3-6 Hz is also a big(p) feature of PD. This may sustain difficulties in handwriting as a symptom. Impaired postural reflexes is also a presenting feature in PD. Patients can easily lose their eternal sleep when pushed slightly, and may need to be caught to keep from falling. These signs can be tried and true by observing the patients walking, getting out of deep chairs, and performing fast repetitive movements. Increased disturbances in cognitive abilities can also fancy evidence of PD. Even with all these signs of PD, it may be present and unknown f... ...ly researched today. Hopefully, the disease will soon be able to be diagnosed too soon enough that its progression can be stopped and be cured. With online research and development, this day may soon come. Works CitedJenner, P. Clues to the mechanism vestigial dopamine cell death -in Parkinsons disease. Journal of Neurology Neurosurqerv, and psychological medicine (1989) 22-28. Korczyr., A. D. Autonomic Nervous System Disturbances in Parkinsons disorder. Advances in Neurology, 53 (1990) 463-68. Kingston, J. W. Current theories on the cause of Parkinsons disease. Journal of Neurology, Neurosurgery, and Psvchiatry, (l989) 13-17. Langston, J. W. and W. C. Koller. The next bourne Presymptomatic detection. Geriatrics, Aug. 1991 5-7. Paulson, G. W. Management of the patient with newly -diagnosed Parkinsons disease. Geriatrics, Feb. 1993 30-40. Parkinsons Disease Essay -- Medical Medical Medicine Essays TreatmentParkinsons DiseaseParkinsons Disease (PD), the shaking palsy first described by James Parkinson in 1817, is a progressive neurodegenerative disorder which affects in upward of 1.5 million Americans. The dis ease begins to occur around age 40 and has incidence with patient age. One survey found that PD may affect 1% of the population over 60. Incidence seems to be more prominent in men, and tends to progress to incapacity and death over one or two decades. Clinical diagnosis of PD is currently solely low-level on the presentation of the symptoms by the patient which reflect a deficiency of striatal dopamine caused by the destruction of the cells in the substantia nigra. Imaging and other testing ground techniques can be used to rule out other disorders, nevertheless are not necessary for the actual diagnosis of PD. The first sign of PA is usually bradykinesia. Movements are usually quite slow. Routine activities may require deliberate planning and thought for execution. Difficulty initiating movements or akinesia, may also be present. Rigidity in the flexors is also present. This is due to an amplify response to normal proprioceptive return from the somatic musculature. A resting apprehension of 3-6 Hz is also a prominent feature of PD. This may cause difficulties in handwriting as a symptom. Impaired postural reflexes is also a presenting feature in PD. Patients can easily lose their remainder when pushed slightly, and may need to be caught to keep from falling. These signs can be tried and true by observing the patients walking, getting out of deep chairs, and performing rapid repetitive movements. Increased disturbances in cognitive abilities can also bespeak evidence of PD. Even with all these signs of PD, it may be present and undiagnosed f... ...ly researched today. Hopefully, the disease will soon be able to be diagnosed early on enough that its progression can be stopped and be cured. With current research and development, this day may soon come. Works CitedJenner, P. Clues to the mechanism central dopamine cell death -in Parkinsons disease. Journal of Neurology Neurosurqerv, and psychiatry (1989) 22-28. Korczyr., A. D. Autonomic Nervous System Disturbances in Parkinsons Disease. Advances in Neurology, 53 (1990) 463-68. Kingston, J. W. Current theories on the cause of Parkinsons disease. Journal of Neurology, Neurosurgery, and Psvchiatry, (l989) 13-17. Langston, J. W. and W. C. Koller. The next barrier Presymptomatic detection. Geriatrics, Aug. 1991 5-7. Paulson, G. W. Management of the patient with newly -diagnosed Parkinsons disease. Geriatrics, Feb. 1993 30-40.